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Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling (microRNA)
PubMed Full text in PMC Similar studies SRA Run Selector
Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling
PubMed Full text in PMC Similar studies
Virus-like vesicles of Kaposi’s Sarcoma-Associated Herpesvirus activate lytic replication through triggering differentiation signaling (mRNA)
Global host gene expression changes in KSHV+ PEL cells upon KSHV reactivation
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Genome-wide mapping of RTA binding sites in KSHV+ PEL cells during KSHV reactivation.
Host gene expression analysis in oral epithelial cells upon KSHV infection
PubMed Full text in PMC Similar studies Analyze with GEO2R
Effect of CTCF and Rad21 knockdown on SLK cells and KSHV gene expression
Effect of CTCF and Rad21 knockdown on cell and KSHV gene expression
XBP-1 transduction of PEL
Up-regulation of ATF4-LAMP3 axis by ORF45 facilitate lytic replication of Kaposi’s sarcoma-associated herpesvirus
Epstein–Barr virus (EBV) Rta-mediated cell cycle arrest enables permissive replication of EBV and Kaposi’s sarcoma-associated herpesvirus in 293 cells
Expression profiling of host genes modulated by Epstein-Barr virus Rta in nasopharyngeal carcinoma cells
Expression profiling of host genes modulated by Epstein-Barr virus (EBV) Rta in HEK293 cells
Transcriptome Analysis of HMGB1 knock-out iSLK BAC16 cell (2)
Transcriptome Analysis of Wild Type and HMGB1 knock-out iSLK BAC16 cells
Transcriptome Analysis of KSHV during de novo primary infection of human B-and endothelial-cells
Transcriptome Analysis of KSHV virions produced from BCBL1 and BAC36 in 293L cells
Transcriptome Analysis of KSHV during de novo primary infection of endothelial (TIVE) cells
Transcriptome Analysis of KSHV during de novo primary infection of human B cells (PBMCs)
Transcriptome Analysis of KSHV during de novo primary infection of human CD14+ cells
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