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Glucocorticoid receptors drive breast cancer cell migration and metabolic reprograming via pSer134-GR-induced expression of PDK4
PubMed Full text in PMC Similar studies Analyze with GEO2R
Glucocorticoid Receptors are Required Effectors of TGFb1-Induced p38 MAPK Signaling to Advanced Cancer Phenotypes in Triple Negative Breast Cancer
PubMed Full text in PMC Similar studies Analyze with GEO2RSRA Run Selector
Role of Serine 134 phosphorylation of the glucocorticoid receptor (GR) in glucocorticoid signaling
PTK6 domain mutants modulates action in Triple-negative breast cancer
PRMT5 and HP1 funtion as coregulators of glucocorticoid receptor in MDA-MB-231 cells
Integrated analysis of DNA methylation, chromatin accessibility, transcription factor binding, and gene expression in large collections of breast cancer cell lines and patient tumors
PubMed Full text in PMC Similar studies
STAT3 and GR cooperate to drive basal-like triple negative breast cancer gene expression and proliferation
PubMed Full text in PMC Similar studies SRA Run Selector
Reduced representation bisulfite sequencing (RRBS) of breast cancer cell lines
RNA-seq of DEX induced and ethanol control Basal-like breast cancer cell lines
Gene expression profiling of MDA_MB_231 cell line in response to treatment with Dexamethasone and SH4-54
Kynurenine 3-monooxygenase upregulates pluripotent genes through β-catenin and promotes triple-negative breast cancer progression
Genome-wide mapping of SRC-3 binding in the liver at CT4.
Coactivator-Dependent Oscillation of Chromatin Accessibility Dictates Circadian Gene Amplitude through REV-ERB Loading
Detection of chromatin accessibility in WT and SRC-2-/- mice in liver
Genome-wide maps of BAF180 and REV-ERBα binding
ChIP-seq and mRNA-seq experiment to find the direct target genes of ATF4 and CHOP
Estrogen Receptor Beta 1: A Potential Therapeutic Target for Triple Negative Breast Cancer
Genomic Regulation of Invasion by STAT3 in Triple Negative Breast Cancer
Genome-wide maps of XBP1 binding sites in different breast cancer cell lines.
Expression data from two breast cancer cell lines
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